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However, previous research in animal models of ASD has shown increased glutamate receptor binding during adolescence instead of childhood, potentially reflecting compensatory mechanisms for earlier synaptic alterations [60]

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3.1.2 Relationship between lipid metabolism and ferroptosis PUFAs, particularly those containing bis-allylic hydrogen atoms such as arachidonate (AA) and adrenate (AdA), are highly susceptible to oxidation by ROS, making them central to ferroptosis (81)